PLASTICIZER DINP DIISONONYL PHTHALATE CAS NO 68515-48-0 KOREA Morocco

  • PLASTICIZER DINP DIISONONYL PHTHALATE CAS NO 68515-48-0 KOREA Morocco
  • PLASTICIZER DINP DIISONONYL PHTHALATE CAS NO 68515-48-0 KOREA Morocco
  • PLASTICIZER DINP DIISONONYL PHTHALATE CAS NO 68515-48-0 KOREA Morocco
  • Is diisononyl phthalate a health hazard?
  • This review of diisononyl phthalate (DINP) is a health hazard assessment only. For this assessment, two key reviews on DINP prepared by the European Chemicals Bureau (ECB, 2003) and the Centre for the Evaluation of Risks to Human Reproduction (CERHR, 2003) were consulted.
  • Is DINP a plasticizer?
  • GoodGuide’s Pollution Scorecard identifies use of DINP as a plasticizer in industrial softeners. CPCat identifies use of DINP in industrial plasticizers. SPIN identifies use of DINP in softeners in Nordic countries. Expected users are based on reporting under GoodGuide’s industrial uses and CPCat’s source (2012 CDR Industrial Sector report).
  • Are DINP phthalates dangerous?
  • Recently, DINP and other related phthalates have received publicity as potential environmental contaminants and raised concern for human health. There are few restrictions on these substances regarding acceptable levels in industrial wastes, liquid effluents, or workplace environments.
  • What CAS number is DINP toxicity?
  • In a 21-d study to assess the toxicity of DINP identified under three CAS numbers, 71549-78-5, 68515-48-0, and 28553-12-0, Fischer Table 5. Acute toxicity of DINP in mammals. F-344 rats were fed diets containing 0%, 0.6%, 1.2%, or 2.5% of the plasticizer.
  • Is phthalate a plasticizer?
  • At that time, di(2-ethylhexyl) phthalate (DEHP) was the predominant plasticizer in PVC articles made for children and in food contact plastics (Canada 1994). In response to human health and safety concerns from regulators and consumer groups, its use in those commercial products was voluntarily reduced and phased out by industry.
  • What is CAS 68515-48-0?
  • For CAS 68515-48-0, an increased occurrence of foetal skeletal variations at 1000 mg/kg bw/d, consisting mainly of rudimentary cervical and accessory 14th ribs (no quantitative data was available), led to a NOAEL for developmental toxicity of 200 mg/kg bw/d. The NOAEL for maternal toxicity was 200 mg/kg bw/d.

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